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See Latest ArticlesInteraction between cytochrome P450 1A2 genetic polymorphism and cigarette smoking on the risk of hepatocellular carcinoma in a Japanese population
Carcinogenesis, 10/08/09
Imaizumi T et al. – Limited epidemiological evidence suggests that genetic polymorphisms of drug–metabolizing enzymes such as cytochrome P450 (CYP), glutathione S–transferase (GST) and N–acetyltransferase (NAT) may be involved in tobacco–related hepatocarcinogenesis. The results suggest that the CYP1A2 –3860G>A polymorphism modifies the smoking–related HCC risk among CLD patients.
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Today in Basic Science/Genetics...keeping you current
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Thrombopoietin in normal and neoplastic stem cell development
Best Practice & Research: Clinical Haematology , 12/07/09
Mechanisms of mutations in myeloproliferative neoplasms
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Ursodeoxycholic acid switches oxaliplatin-induced necrosis to apoptosis by inhibiting reactive oxygen species production and activating p53-caspase 8 pathway in HepG2 hepatocellular carcinoma
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Today in Hepatobiliary/Pancreas...keeping you current
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PBK/TOPK in the differential diagnosis of cholangiocarcinoma from hepatocellular carcinoma and its involvement in prognosis of human cholangiocarcinoma
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Hepatic Arterial Embolization and Chemoembolization for Imatinib-Resistant Gastrointestinal Stromal Tumors
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