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Chronic cigarette smoke extract treatment selects for apoptotic dysfunction and mitochondrial mutations in minimally transformed oral keratinocytes
International Journal of Cancer, 09/29/09
Chang SS et al. – Chronic tobacco exposure induces carcinogenesis via selection of apoptosis resistance and mitochondrial mutation in addition to previously known genotoxic effects that were found by acute treatments. Chronic models of tobacco exposure on upper aerodigestive epithelia may be more insightful than models of acute exposure in studying head and neck carcinogenesis.
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Knockdown of peroxisome proliferator-activated receptor-beta induces less differentiation and enhances cell–fibronectin adhesion of colon cancer cells
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Today in Head and Neck...keeping you current
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Paraneoplastic syndromes in patients with oral cancer
Oral Oncology , 11/25/09
Early radiation-induced laryngeal malignant fibrous histiocytoma (MFH)
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