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Wang X et al. – The results demonstrated that 28 k m/z peak was apolipoprotein A–I (ApoA–I) and its identity was further validated by immunodepletion and Western blotting analysis. Subsequently, it was inspiring found that the decreased level of ApoA–I analysed by enzyme linked immunosorbent assay was consistent with SELDI–TOF–MS analysis. Therefore, it suggested that ApoA–I could be a potential useful biomarker for CC.

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