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See Latest ArticlesReactivating HIF prolyl hydroxylases under hypoxia results in metabolic catastrophe and cell death
Oncogene, 09/03/09
Tennant DA et al. – Cells exposed to low–oxygen conditions (hypoxia) alter their metabolism to survive. This response, although vital during development and high–altitude survival, is now known to be a major factor in the selection of cells with a transformed metabolic phenotype during tumorigenesis.
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The prolyl-hydroxylase EGLN3 and not EGLN1 is inactivated by methylation in plasma cell neoplasia
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Today in Basic Science/Genetics...keeping you current
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Expression of p16 in benign and malignant cystic squamous lesions of the neck
Human Pathology, 12/19/09
Correlation of overexpression of HMGA1 and HMGA2 with poor tumor differentiation, invasion, and proliferation associated with let-7 down-regulation in retinoblastomas
Human Pathology, 12/19/09
Beclin 1 and LC3 autophagic gene expression in cutaneous melanocytic lesions
Human Pathology, 12/19/09
Today in Carcinogenesis...keeping you current
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Differential effects of reactive nitrogen species on DNA base excision repair initiated by the alkyladenine DNA glycosylase
Carcinogenesis, 12/19/09
Aggressive inherited and sporadic medullary thyroid carcinomas display similar oncogenic pathways
Endocrine-Related Cancer, 12/08/09
Activation of the hedgehog-signaling pathway in human cancer and the clinical implications
Oncogene, 11/30/09
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