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See Latest ArticlesProlactin confers resistance against cisplatin in breast cancer cells by activating glutathione-S-transferase
Carcinogenesis, 07/30/09
LaPensee EW et al. – The data provide a rational explanation for the ineffectiveness of cisplatin in breast cancer, which is characterized by high expression of both PRL and its receptor. Suppression of PRL production or blockade of its actions should benefit patients undergoing chemotherapy by allowing for lower drug doses and expanded drug options.
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Today in Basic Science/Genetics...keeping you current
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Target genes suitable for silencing approaches and protein product interference in ovarian epithelial cancer
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Dysregulation of Platelet-Derived Growth Factor beta-Receptor Expression by deltaNp73 in Neuroblastoma
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Today in Breast...keeping you current
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Bevacizumab and breast cancer: current therapeutic progress and future perspectives
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Today in Carcinogenesis...keeping you current
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Activation of the hedgehog-signaling pathway in human cancer and the clinical implications
Oncogene, 11/30/09
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