Heart Disease Journals

Cardiology

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Your Article Summary

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Ares–Carrasco S et al. – Fibrosis and apoptosis are features of long–term myocardial damage in experimental DM1. Associated hypertension does not induce additional changes. Myocardial inflammation is present in hypertension and short–term DM1, but is not a key feature in long–term DM1. Local reduction of pro–inflammatory factors and expression of anti–inflammatory and anti–oxidant molecules may underlie this effect.

Exclusive Author Commentary
Óscar Lorenzo, 10/19/09

Anti-inflammatory and/or anti-oxidant specific molecules may be used as a therapy for damaged hearts from diabetic and hypertensive patients.

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