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See Latest ArticlesRegional Distribution of Mitochondrial Dysfunction and Apoptotic Remodeling in Pacing-Induced Heart Failure
Journal of Cardiac Failure, 10/06/09
Marín–García J et al. – Abnormal activity of several mitochondrial enzymes and increased apoptogenic pathway appear to be mediated, at least in part, by an orchestrated shift in expression (both nuclear and mitochondrial DNA) of respiratory chain subunits, mitochondrial bioenergetic enzymes (eg, mitochondrial creatine kinase), global transcription factor, and apoptotic proteins (eg, p53, p21) with distinct differences in their regional distribution and in the subpopulations of mitochondria affected.
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Genotype-Phenotype Aspects of Type 2 Long QT Syndrome
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Today in Heart Failure...keeping you current
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