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See Latest ArticlesHDL3-Mediated Inactivation of LDL-Associated Phospholipid Hydroperoxides Is Determined by the Redox Status of Apolipoprotein A-I and HDL Particle Surface Lipid Rigidity. Relevance to Inflammation and Atherogenesis
Arteriosclerosis, Thrombosis, and Vascular Biology, 09/18/09
Zerrad–Saadi A et al. – The redox status of apoAI and surface lipid rigidity represent major determinants of the potent HDL3–mediated protection of LDL against free radical–induced oxidation. Initial transfer of PCOOH to HDL3 is modulated by the surface rigidity of HDL3 particles with subsequent reduction of PCOOH to PCOH by methionine residues of apoAI.
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