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See Latest ArticlesAlpha-1-Syntrophin Mutations Identified in Sudden Infant Death Syndrome Cause an Increase in Late Cardiac Sodium Current
Circulation: Arrhythmia and Electrophysiology, 09/22/09
Cheng J et al. – Abnormal biophysical phenotypes implicate mutations in SNTA1 as a novel pathogenic mechanism for the subset of channelopathic SIDS. Functional studies are essential to distinguish pathogenic perturbations in channel interacting proteins like alpha–1–syntrophin from similarly rare but innocuous ones.
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Management of apnoea and bradycardia in neonates
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A multicentric study of disease-related stress, and perceived vulnerability, in parents of children with congenital cardiac disease
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