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See Latest ArticlesLack of Cardiac Fibrosis in a New Model of High Prorenin Hyperaldosteronism
Heart And Circulatory Physiology, 09/15/09
Peters J et al. – Prorenin and aldosterone alone are not sufficient to induce considerable cardiac fibrosis in the absence of sodium load.
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Reduced expression of the sodium-calcium exchanger in adult cardiomyocytes via adenovirally delivered shrna results in resistance to simulated ischemic injury
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