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See Latest ArticlesExpression of constitutively active cGMP-dependent protein kinase inhibits glucose-induced vascular smooth muscle cell proliferation
Heart And Circulatory Physiology, 08/31/09
Wang S et al. – The data demonstrate that restoring high glucose–mediated decrease in PKG activity in VSMCs inhibits glucose–induced abnormal VSMCs proliferation occurring upstream of Rb phosphorylation. This work provides the first direct evidence linking decreased PKG activity to high glucose–induced proliferation and cell cycle progression in VSMCs, suggesting that strategies to increase PKG activity might be useful in preventing abnormal VSMCs proliferation in diabetic patients and might provide treatments for diabetes–associated proliferative vascular diseases.
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