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See Latest ArticlesGPD1L links redox state to cardiac excitability by PKC-dependent phosphorylation of the NA channel SCN5A
Heart And Circulatory Physiology, 08/14/09
Valdivia CR et al. – The results show a function for GPD1L in cell physiology, and a mechanism linking mutations in GPD1L to sudden cardiac arrest. Because the enzymatic step catalyzed by GPD1L depends upon nicotinamide adenine dinucleotide (NAD), this GPD1L pathway links the metabolic state of the cell to INa and excitability and may be important more generally in cardiac ischemia and heart failure.
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