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See Latest ArticlesVascular Smooth Muscle Modulates Endothelial Control of Vasoreactivity via Reactive Oxygen Species Production through Myoendothelial Communications
PLoS ONE - Cardiovascular Disorders, 07/31/09
Billaud M et al. – The data demonstrate that agonist–induced ROS production in SMC targeting EC via myoendothelial gap junctions reduces endothelial NO–dependent control of pulmonary vasoreactivity. Such SMC modulation of endothelial control may represent a signaling pathway controlling vasoreactivity under not only physiological but also pathological conditions that often implicate excessive ROS production.
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