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Overexpression of Hsp20 prevents endotoxin-induced myocardial dysfunction and apoptosis via inhibition of NF-kB activation
Journal of Molecular and Cellular Cardiology, 08/04/09
Wang X et al. – The increases in Hsp20 levels can protect against LPS–induced cardiac apoptosis and dysfunction, associated with inhibition of NF–kB activity, suggesting that Hsp20 may be a new therapeutic agent for the treatment of sepsis.
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