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See Latest ArticlesIncreased InsP3Rs in the junctional sarcoplasmic reticulum augment Ca2+ transients and arrhythmias associated with cardiac hypertrophy
Proceedings of the National Academy of Sciences of the United States of America, 07/17/09
Harzheim D et al. - The data establish that increased InsP3R expression is a general mechanism that underlies remodeling of Ca2+ signaling during heart disease, and in particular, in triggering ventricular arrhythmia during hypertrophy.
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