Your Article Summary
High glucose sensitizes adult cardiomyocytes to ischaemia/reperfusion injury through nitrative thioredoxin inactivation
Cardiovascular Research, 06/30/09
Luan R et al. - High glucose sensitized cardiomyocytes to ischaemia/reperfusion injury through nitrative Trx-1 inactivation. Interventions restoring Trx-1 activity in the diabetic heart may represent novel therapies attenuating cardiac injury in diabetic patients.
Related Articles
Hypersensitivity of excitation-contraction coupling in dystrophic cardiomyocytes
Heart And Circulatory Physiology, 10/14/09
Relevance Score: 87%
Human Amniotic Mesenchymal Cells Have Some Characteristics of Cardiomyocytes
Transplantation, 10/06/09
Relevance Score: 87%
Dok-5 is involved in cardiomyocyte differentiation through PKB/FOXO3a pathway
Journal of Molecular and Cellular Cardiology, 11/10/09
Relevance Score: 84%
Cell Cycle Re-Entry and Mitochondrial Defects in Myc-Mediated Hypertrophic Cardiomyopathy and Heart Failure
PLoS ONE - Cardiovascular Disorders, 10/02/09
Relevance Score: 84%
Role of Ca2+/Calmodulin–Stimulated Cyclic Nucleotide Phosphodiesterase 1 in Mediating Cardiomyocyte Hypertrophy
Circulation Research, 10/06/09
Relevance Score: 83%
Today in Myocardium/Pericardium...keeping you current
Receive free subspecialty "5-minute updates" via email
Association of Hospital Primary Angioplasty Volume in ST-Segment Elevation Myocardial Infarction With Quality and Outcomes
JAMA, 11/25/09
Mode of arrival does not predict myocardial infarction in patients who present to the ED with chest pain
International Journal of Emergency Medicine, 11/25/09
Increased Carotid Intima Media Thickness as a Predictor of the Presence and Extent of Abnormal Myocardial Perfusion in Type 2 Diabetes Mellitus
Diabetes Care, 11/23/09

See Latest Articles