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See Latest ArticlesFeatures of cardiac allograft coronary endothelial dysfunction
The American Journal of Cardiology, 04/15/09
Raichlin E et al. – Cardiac allograft epicardial coronary endothelial function was abnormal and may be related to an impaired endogenous NO synthetase pathway and reduced endothelial nitric oxide production in transplant recipients.
Methods- Study of features and mechanism of cardiac allograft coronary endothelial dysfunction
- Assessment of coronary blood flow and epicardial coronary artery diameter response to intracoronary acetylcholine and NG-monomethyl-l-arginine
- Review of 19 cyclosporine-treated heart transplant recipients with normal coronary angiograms (3.8 ± 2.3 yrs post-transplantation)
- Comparison with 19 age-, gender-, and cardiovascular risk factor–matched nontransplantation controls with normal coronary angiograms
- More epicardial vasoconstriction in response to intracoronary acetylcholine for heart transplant recipients
- Microvascular endothelial function similar between groups
- Coronary flow reserve in response to adenosine higher in transplant pts
- Effect of NG-monomethyl-l-arginine (64 micromol/min) on coronary blood flow, coronary artery diameter, and coronary vascular resistance significantly attenuated in transplant pts vs controls
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