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See Latest ArticlesPepsin as a causal agent of inflammation during nonacidic reflux
Otolaryngology Head and Neck Surgery, 10/28/09
Samuels TL et al. – Exposure of hypopharyngeal cells to pepsin in a nonacidic environment induces the expression of several pro–inflammatory cytokines and receptors, including those known to be involved in inflammation of esophageal epithelium in response to reflux and which contribute to the pathophysiology of reflux esophagitis. These data indicate that refluxed pepsin may contribute to laryngeal inflammation associated with nonacidic gastric reflux, including that experienced by patients despite maximal acid suppression therapy.
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