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See Latest ArticlesRole of LPS/CD14/TLR4-mediated inflammation in necrotizing enterocolitis: Pathogenesis and therapeutic implications
World Journal of Gastroenterology, 10/20/09
Chan K et al. – LPS and distension are required to produce the histological and inflammatory features of NEC. A potential treatment option is blocking LPS activation and leukocyte infiltration.
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Today in Basic Science/Genetics...keeping you current
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Epithelial apoptosis: Cause or consequence of ulcerative colitis
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Today in GI Infectious Disease...keeping you current
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An ex-vivo Human Intestinal Model to Study Entamoeba histolytica Pathogenesis
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