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See Latest ArticlesInsulin, leptin and reward
Trends in Endocrinology and Metabolism, 11/02/09
Davis JF et al. – Feeding for pleasure, or “non–homeostatic feeding”, potentially contributes to the rapid development of obesity worldwide. Obesity is associated with an imbalance of regulatory hormones which normally act to maintain stable energy balance and body weight. The adiposity hormones insulin and leptin are two such signals elevated in obesity with the capacity to dampen feeding behavior through their action on hypothalamic circuits which regulate appetite and metabolism. Recent evidence suggests that both hormones achieve this degree of regulation by inhibiting the rewarding aspects of feeding behavior, perhaps by signaling within midbrain reward circuits. This review describes the capacity of both insulin and leptin to regulate reward–related behavior.
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An integrative view of obesity
American Journal of Clinical Nutrition, 11/19/09
Marine Omega-3 Fatty Acid Intake: Associations with Cardiometabolic Risk and Response to Weight Loss Intervention in The Look AHEAD Study
Diabetes Care, 11/05/09
Comparison of Combined Bupropion and Naltrexone Therapy for Obesity with Monotherapy and Placebo
Journal of Clinical Endocrinology and Metabolism, 10/29/09
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