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Z and Mmalton-1-antitrypsin deficiency-associated hepatocellular carcinoma: a genetic study
Liver International, 10/08/09

Francalanci P et al. – The authors findings indicate that both variants of HCC arising in AAT deficiency (Z and Mmalton) do not accumulate the mutant protein and the different phenotypic appearance of hepatocytes is not the result of a retromutation during neoplastic transformation, but other mechanisms should be investigated.

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