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McCallum RW et al. – The authors conclude that data suggest that the symptomatic improvement achieved by GES in gastroparesis is best explained by activation of vagal afferent pathways to influence CNS control mechanisms for nausea and vomiting accompanied by enhanced vagal efferent autonomic function and decreased gastric sensitivity to volume distention which enhances postprandial gastric accommodation.

Today in Basic Science/Genetics...keeping you current

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Today in GI Motility...keeping you current

ERCP-induced and non-ERCP-induced acute pancreatitis: Two distinct clinical entities with different outcomes in mild and severe form
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An Antireflux Anastomosis Following Esophagectomy: A Randomized Controlled Trial
Journal of Gastrointestinal Surgery, 12/17/09


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