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See Latest ArticlesElevated insular glutamate in fibromyalgia is associated with experimental pain
Arthritis & Rheumatism, 10/02/09
Harris RE et al. – Central pain augmentation resulting from enhanced excitatory and/or decreased inhibitory neurotransmission is a proposed mechanism underlying the pathophysiology of functional pain syndromes such as fibromyalgia (FM)...Enhanced glutamatergic neurotransmission resulting from higher concentrations of Glu within the posterior insula may play a role in the pathophysiology of FM and other central pain augmentation syndromes.
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