ENT Medical News about FOXI1

Become a Member Today!
Register

Most Viewed Abstracts

1. Report Shows Shift in Starting Salaries for Physicians

2. 2008 Exclusive Survey—Earnings: Good news for primary care income

3. Medicare pay-for-reporting effort draws fire from frustrated doctors

4. Debunking Myths in the US Healthcare System

5. Appropriate Antibiotic Use for Acute Otitis Media

Your Article Summary

See Latest Articles

(Click the title below to leave the MDLinx Network and go to the Journal's Website)

Phenotypic Analyses and Mutation Screening of the SLC26A4 and FOXI1 Genes in 101 Taiwanese Families with Bilateral Nonsyndromic Enlarged Vestibular Aqueduct or Pendred Syndrome
Audiology & Neuro-Otology, 08/03/09

Wu CC et al. – In most Taiwanese families nonsyndromic EVA or Pendred syndrome might not result from aberrance in the transcriptional control of SLC26A4 by FOXI1. Meanwhile, exploration of undetected mutations in the SLC26A4 noncoding regions revealed 9 divergent haplotypes among the 21 no–mutation–detected SLC26A4 alleles of the c.919–2A>G heterozygotes, indicating that there might be no common and predominant mutations in the SLC26A4 introns.

<< Previous
Propranolol for airway hemangiomas: Case report of novel...

Next >>
Absence of Simian virus 40, BK, and JC polyomavirus DNA in...