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See Latest ArticlesThe genetics of familial glucocorticoid deficiency
Best Practice & Research Clinical Endocrinology & Metabolism, 06/10/09
Clark AJL et al. - Patients present in early life with low or undetectable cortisol and – because of the failure of the negative feedback loop to the pituitary and hypothalamus – grossly elevated ACTH levels. About half of all cases result from mutations in the ACTH receptor (melanocortin 2 receptor) or from mutations in the melanocortin 2 receptor accessory protein (MRAP), but other genetic causes of this potentially lethal disorder remain to be discovered.
Today in Adrenal Glands...keeping you current
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ACTH-Independent Cushing's Syndrome with Bilateral Micronodular Adrenal Hyperplasia and Ectopic Adrenocortical Adenoma
Journal of Clinical Endocrinology and Metabolism, 11/25/09
Isocitrate Dehydrogenase Mutations Are Rare in Pheochromocytomas and Paragangliomas
Journal of Clinical Endocrinology and Metabolism, 11/25/09
Association of the M1V PRKAR1A Mutation with Primary Pigmented Nodular Adrenocortical Disease in Two Large Families
Journal of Clinical Endocrinology and Metabolism, 11/25/09
Today in Basic Science/Genetics...keeping you current
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Examination of all type 2 diabetes GWAS loci reveals HHEX-IDE as a locus influencing pediatric BMI
Diabetes, 11/30/09
Reduced plasma levels of glucagon-like peptide-1 in elderly men are associated with impaired glucose tolerance but not with coronary heart disease
Diabetologia - Clinical and Experimental Diabetes and Metabolism, 11/30/09
Association of the PPARGC1A Gene Polymorphism With Diabetic Nephropathy in an Asian Indian Population (CURES-41)
Metabolic Syndrome and Related Disorders, 11/30/09
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