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polycystic ovary syndrome Article Summary

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Impaired insulin activation and dephosphorylation of glycogen synthase in skeletal muscle of women with in polycystic ovary syndrome is reversed by pioglitazone treatment
Journal of Clinical Endocrinology and Metabolism, 06/11/08
Print     Email This Article     Save in My Library   Free Abstract
Glintborg D et al. - Impaired insulin activation of GS including absent dephosphorylation at sites 2+2a contributes to insulin resistance in skeletal muscle in PCOS. The ability of pioglitazone to enhance insulin sensitivity, in part, involves improved insulin action on GS activity and dephosphorylation at NH2-terminal sites.
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