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PPAR[alpha] mediates the hypolipidemic action of fibrates by antagonizing FoxO1
Endocrinology and Metabolism, 02/21/07
Print     Email This Article     Save in My Library   Free Abstract
Shen Qu., et al. – High-fructose consumption is associated with insulin resistance and diabetic dyslipidemia, but the underlying mechanism is unclear. We show in hamsters that high-fructose feeding stimulated forkhead box O1 (FoxO1) production and promoted its nuclear redistribution in liver, correlating with augmented apolipoprotein C-III (apoC-III) production and impaired triglyceride metabolism

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