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See Latest ArticlesDelay of Lps-Induced Acute Lung Injury Resolution By Soluble Immune Complexes Is Neutrophil Dependent
Shock, 08/24/09
Wu CL et al. – Excessive amount of SICX in lung can act through FcgammaRs to protect bronchoalveolar lavage fluid neutrophils from apoptosis that eventually lead to delayed resolution of ALI caused by LPS. Blockade of SICX engagement of CD32 and CD64 (with decoy antibodies) could interrupt SICX–mediated protection of neutrophils and protect lung from LPS–induced injury. The decoy antibodies may therefore have therapeutic utility in ALI.
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