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See Latest ArticlesThe cell cycle and acute kidney injury
Kidney International, 06/22/09
Price PM et al. - Acute kidney injury (AKI) activates pathways of cell death and cell proliferation. Although seemingly discrete and unrelated mechanisms, these pathways can now be shown to be connected and even to be controlled by similar pathways. The dependence of the severity of renal-cell injury on cell cycle pathways can be used to control and perhaps to prevent acute kidney injury.
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Migraine Chronification - Concept and Risk Factors
Discovery Medicine, 10/27/09
Inhibition of NF-kB ameliorates sepsis-induced downregulation of aquaporin-2 / V2 receptor expression and acute renal failure in vivo
American Journal of Physiology: Renal Physiology, 10/15/09
Elevated Saliva Calcitonin Gene-Related Peptide Levels During Acute Migraine Predict Therapeutic Response to Rizatriptan
Headache: The Journal of Head and Face Pain, 10/14/09
Today in Urogenital/Nephrology...keeping you current
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Alpha blockers prior to removal of a catheter for acute urinary retention in adult men
Cochrane Reviews, 10/19/09
Acute phosphate nephropathy
Current Opinion in Nephrology and Hypertension, 10/15/09
Non-dialytic management of sepsis-induced acute kidney injury
Saudi Journal of Kidney Diseases and Transplantation, 11/02/09
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