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Livedoid vasculiltis associated with a double heterozygous Factor V Leiden and prothrombin G20210A gene mutations
Clinical and Experimental Dermatology, 10/08/09

Dalle S et al. – Livedoid vasculitis (LV) may be an isolated condition or associated with an underlying systemic disease, including coagulation disorders, periarteritis nodosa and systemic autoimmune diseases. The classic histological features of LV (fibrin deposition on dermal vessels and the absence of important perivascular infiltrate or leucocytoclasia) argue against a primary vasculitis and is more in keeping with a thrombotic process. Factor V Leiden mutations have rarely been reported in association with LV.

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The prevalence of factor V Leiden (G1691A), prothrombin G20210A and methylenetetrahydrofolate reductase C677T mutations in Jordanian patients with [beta]-thalassemia major
Blood Coagulation & Fibrinolysis, 11/03/09 Relevance Score: 84%

Impact of infusion speed on the safety and effectiveness of prothrombin complex concentrate
Annals of Hematology, 10/02/09 Relevance Score: 84%

Prothrombin fragment 1+2 is associated with intima media thickness of the carotid artery in patients with myocardial infarction
Thrombosis Research, 10/20/09 Relevance Score: 83%

Recombinant activated factor VII for a patient with factor VII deficiency undergoing urgent intracerebral haematoma evacuation with underlying cavernous angioma
British Journal of Anesthesia, 10/26/09 Relevance Score: 80%

Prevalence of factor V Leiden and prothrombin G20210A mutation in a large French population selected for nonthrombotic history: geographical and age distribution
Blood Coagulation & Fibrinolysis, 10/05/09 Relevance Score: 80%