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Propofol attenuates angiotensin II-induced vasoconstriction by inhibiting Ca2+-dependent and PKC-mediated Ca2+ sensitization mechanisms
Journal of Anesthesia,

Kuriyama T et al. – The results suggest that the inhibitory effect of propofol on Angiotensin II (Ang II)–induced vascular contraction is mediated by the attenuation of a Ca2+–dependent pathway and Ca2+ sensitivity through the protein kinase C (PKC) signaling pathway.

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