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See Latest ArticlesElucidating the role of reversible protein phosphorylation in sepsis-induced myocardial dysfunction
Shock, 06/25/09
Lorts A et al. - The authors hypothesize that key signaling pathways that control myocardial calcium flux, regulated to key kinases and phosphatases, influence myocyte contractility in sepsis. Thus, they review the data relevant to the sepsis-induced intracellular alterations in calcium flux in the cardiomyocyte, with an emphasis on changes in the phosphorylation state of the contractile proteins regulated by the balance between kinases and phosphatases.
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