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See Latest ArticlesEffect of SOD-1 over-expression on myocardial function during resuscitated murine septic shock
Intensive Care Medicine, 10/17/08
Baumgart K et al. - The authors conclude that neither hetero- nor homozygous SOD-1 over-expression caused a sustained improvement of the sepsis-related impairment of myocardial norepinephrine responsiveness, possibly due to the lacking increase of the tissue catalase and the mitochondrial SOD activity as well as the ongoing i.v. noradrenaline.
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