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See Latest ArticlesThe role of redox-dependent mechanisms in the downregulation of ligand-induced Toll-like receptors 7, 8 and 4-mediated HIF-1 prolyl hydroxylation
Immunology & Cell Biology, 10/26/09
Nicholas SA et al. – RNS and p38 MAP kinase (known to contribute to LPS–induced TLR4–dependent accumulation of HIF–1 protein) do not affect HIF–1 prolyl hydroxylation. In both cases, downregulation of HIF–1 prolyl hydroxylation correlates with a decrease in intracellular iron (II).
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