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See Latest Articles2-Deoxy-d-ribose induces cellular damage by increasing oxidative stress and protein glycation in a pancreatic beta-cell line
Metabolism-Clinical and Experimental, 10/13/09
Koh G et al. – On incubation with albumin, dRib increased dicarbonyl and advanced glycation end product formation. Aminoguanidine and pyridoxamine significantly decreased the dicarbonyl and advanced glycation end product augmentations. These results suggest that both oxidative stress and protein glycation are important mechanisms of dRib–induced damage in a pancreatic beta–cell line.
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