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See Latest ArticlesEndotoxin tolerance: new mechanisms, molecules and clinical significance
Trends in Immunology, 10/09/09
Biswas SK et al. – The recent appreciation of inflammation as a self–regulating process, the relative contribution of MyD88 versus TRIF signaling pathways in inducing activation or tolerance, plasticity of NF–kB function and the role of chromatin modification and microRNAs in LPS–induced gene reprogramming urges a re–evaluation of endotoxin tolerance. This review integrates these new findings into an up–to–date account of endotoxin tolerance, its molecular basis and clinical implications in different pathologies.
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