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Yang H et al. – RelB–silenced DCs were able to create a particular cytokine environment that was absent of inflammatory cytokines. RelB–silenced DCs provide a practical means to normalize the differentiation of the four T–cell subsets (Th17, Th1, Th2, and Treg) in vivo, and thus possess therapeutic potential in Th1/Th17–dominant autoimmune disorders such as myasthenia gravis.


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