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Stable silencing of the glucocorticoid receptor in myelin-specific T effector cells by retroviral delivery of shRNA: Insight into neuroinflammatory disease
European Journal of Immunology, 08/17/09
Tischner D et al. – This highlights an important role of other cell types and bystander T cells as targets of glucocorticoid therapy. Collectively, this approach provides a simple tool for stable and efficient gene silencing in T effector cells, which should help to better understand brain autoimmune pathophysiology.
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Helicobacter pylori Stimulates Dendritic Cells to Induce IL-17 Expression From CD4+ T lymphocytes
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CXCL10 production by human monocytes in response to Leishmania braziliensis infection
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Coactivation of Syk Kinase and MyD88 Adaptor Protein Pathways by Bacteria Promotes Regulatory Properties of Neutrophils
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Clonally expanded plasma cells in the cerebrospinal fluid of patients with central nervous system autoimmune demyelination produce “oligoclonal bands”
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Update in treatment options for psoriatic arthritis
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