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Interplay between signaling via the formyl peptide receptor (FPR) and chemokine receptor 3 (CCR3) in human eosinophils
Journal of Leukocyte Biology, 08/07/09
Svensson L et al. – Pharmacologic inhibition of signal transduction molecules showed that the release of free oxygen radicals in response to eotaxin–1 compared with fMLP is relatively more dependent on the p38 MAPK pathway.
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Today in Hematology/Oncology...keeping you current
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Can antigen-specific regulatory T cells protect against graft vs. host disease and spare anti-malignancy alloresponse
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