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See Latest ArticlesAntiphospholipid Antibodies Induce a Pro-Inflammatory Response in First Trimester Trophoblast Via the TLR4/MyD88 Pathway
American Journal of Reproductive Immunology, 07/14/09
Mulla MJ et al. - These findings demonstrate that aPL triggers a placental inflammatory response via the TLR-4/MyD88 pathway, which in turn compromises trophoblast survival. Thus, the TLR-4/MyD88 pathway may provide a new therapeutic target to improve pregnancy outcome in antiphospholipid syndrome patients.
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Today in Maternal Fetal Medicine...keeping you current
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A role for IL-17 in induction of an inflammation at the fetomaternal interface in preterm labour
Journal of Reproductive Immunology, 12/04/09
Spermadhesin PSP-I/PSP-II heterodimer induces migration of polymorphonuclear neutrophils into the uterine cavity of the sow
Journal of Reproductive Immunology, 12/04/09
Allergic women show reduced T helper type 1 alloresponses to fetal human leucocyte antigen mismatch during pregnancy
Clinical and Experimental Immunology, 12/03/09
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