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See Latest ArticlesNeuroprotective effects of the complement terminal pathway during demyelination: Implications for oligodendrocyte survival
Journal of Neuroimmunology, 07/10/09
Tegla CA et al. - Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system that is mediated by activated lymphocytes, macrophages/microglia, and complement. In MS, the myelin-forming oligodendrocytes (OLGs) are the targets of the immune attack. The findings indicate that the activation of complement and C5b-9 assembly can also have protective roles during demyelination.
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Today in Basic Science/Genetics...keeping you current
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Recipient NOD2/CARD15 status affects cellular infiltrates in human intestinal graft-versus-host disease
Clinical and Experimental Immunology, 12/04/09
Milder clinical hyperimmunoglobulin E syndrome phenotype is associated with partial interleukin-17 deficiency
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Effects of PARP-1 deficiency on airway inflammatory cell recruitment in response to LPS or TNF: differential effects on CXCR2 ligands and Duffy antigen receptor for chemokines
Journal of Leukocyte Biology, 12/04/09
Today in Neurologic/Ophthalmic...keeping you current
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Retinal astrocytes respond to IL-17 differently than retinal pigment epithelial cells
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Limited Role of CD4+Foxp3+ Regulatory T Cells in the Control of Experimental Cerebral Malaria
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Tyrosine Kinase 2 Plays Critical Roles in the Pathogenic CD4 T Cell Responses for the Development of Experimental Autoimmune Encephalomyelitis
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