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See Latest ArticlesDefects along the TH17 differentiation pathway underlie genetically distinct forms of the hyper IgE syndrome
Journal of Allergy and Clinical Immunology, 07/28/09
Khatib SA et al. – In this cohort of Turkish children with HIES, a majority had normal STAT3, implicating other targets in disease pathogenesis. Impaired TH17 responses were evident irrespective of the STAT3 mutation status, indicating that different genetic forms of HIES share a common functional outcome.
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