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Chen C et al. - The study revealed the differential regulatory roles and the novel mechanism of action of COP-I chiefly responsible for its treatment efficacy in experimental autoimmune encephalomyelitis and multiple sclerosis.

Today in Rheumatic/Autoimmune...keeping you current

Inhibition of clonal expansion by Foxp3 expression as a mechanism of controlled T-cell responses and autoimmune disease
European Journal of Immunology, 12/07/09

Th1, Th17, and Th9 Effector Cells Induce Experimental Autoimmune Encephalomyelitis with Different Pathological Phenotypes
Journal of Immunology, 12/07/09

Vasculitis
Journal of Allergy and Clinical Immunology, 12/04/09