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Postnatal adrenalectomy impairs urinary concentrating ability by increased COX-2 and leads to renal medullary injury
Renal Physiology, 08/31/07
Stubbe, J., et al. - It is concluded that mineralocorticoid is required for normal postnatal development of the renal medulla. COX-2 contributes to impaired urine-concentrating ability, NaCl loss, and extracellular volume depletion in postnatal mineralocorticoid deficiency
Today in Basic Science/Genetics...keeping you current
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Paracrine/endocrine mechanism of stem cells on kidney repair: role of microvesicle-mediated transfer of genetic information
Current Opinion in Nephrology and Hypertension, 12/17/09
Susceptibility genes in common complex kidney disease
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The Changing Phenotype of Academic Nephrology— A Future at Risk
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Increased Urinary Angiotensinogen Is Precedent to Increased Urinary Albumin in Patients With Type 1 Diabetes
The American Journal of the Medical Sciences, 12/17/09
Paracrine/endocrine mechanism of stem cells on kidney repair: role of microvesicle-mediated transfer of genetic information
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The renal vascular response to diabetes
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Angiotensin Receptor Blocker Reduces Proteinuria Independently of Blood Pressure in Children Already Treated with Angiotensin-Converting Enzyme Inhibitors
Kidney and Blood Pressure Research, 12/17/09
The impact of the National Patient Safety Agency intravenous fluid alert on iatrogenic hyponatraemia in children
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Development of renal and iliac aneurysms in a child with generalized infantile myofibromatosis
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