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See Latest ArticlesIncreased Expression of Visfatin in Macrophages of Human Unstable Carotid and Coronary Atherosclerosis
Circulation, 03/05/07
Tuva B. Dahl, MSc., et al. – Although the participation of inflammation in atherogenesis is widely recognized, the identification of the different components has not been clarified. In particular, the role of inflammation in plaque destabilization is not fully understood...Conclusion: Our findings suggest that visfatin should be regarded as an inflammatory mediator, localized to foam cell macrophages within unstable atherosclerotic lesions, that potentially plays a role in plaque destabilization
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Today in Cardiology...keeping you current
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Increased NT-pro-B-type natriuretic peptide independently predicts outcome following catheter ablation of atrial fibrillation
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Today in Vascular Medicine...keeping you current
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GNB3 C825T Polymorphism and Elevated Blood Pressure
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