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See Latest ArticlesAdenosine A1 but Not A2a Receptor Agonist Reduces Hyperalgesia Caused by a Surgical Incision in Rats: A Pertussis Toxin-sensitive G Protein-dependent Process
Anesthesiology, 11/01/07
Zahn, P.K., et al. - Conclusions: Spinal A1Rs but not A2aRs play an important role in the maintenance of nonevoked and evoked pain behaviors after an incision. Furthermore, A1R-induced spinal antinociception is mediated by interactions with pertussis toxin-sensitive G proteins. In addition, the opening of adenosine triphosphate-sensitive K channels but not of calcium-activated potassium channels and voltage-gated Kv1.3 or Kv1.6 channels contribute to the antinociceptive effect of A1R agonists
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